By P. D. Aplan, M. Stanulla (auth.), Prof. Dr. T. Büchner, Prof. Dr. med. W. Hiddemann, Prof. Dr. B. Wörmann, Prof. Dr. G. Schellong, Prof. Dr. J. Ritter, Prof. Dr. U. Creutzig (eds.)
Since 1987 Acute Leukemias (AL) has frequently mentioned at the cutting-edge within the quickly transforming into, winning and interesting box of biology and administration of those illnesses. during this quantity huge multicenter medical trials back shape the cast foundation for the research of leukemic mobilephone biology, the detection of biologic subgroups, their differential reaction to replacement therapy ideas and the extra improvement of remedy. specifically, this quantity addresses mechanisms of chromosomal translocations and transcription elements, risk-adapted remedy concepts, differentiation treatment, secondary AL, targeted facets in older sufferers, and adoptive immunotherapy. along with the antileukemic ways, supportive remedy with new antimicrobial components and development elements is up-to-date. As a discussion board of globally actions within the box of AL this quantity includes either exhaustive overviews on significant medical concerns and initial information and hypotheses now not formerly published.
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Additional info for Acute Leukemias VIII: Prognostic Factors and Treatment Strategies
9 Thirman MJ, and Larson RA (1996). Therapy-related myeloid leukemia . Hematology Oncology Clinics of North America 10,293-320. Tycko B,and Sklar J (1990). Chromosomal translocations in lymphoid neoplasia: a reappr aisal of the recombinase model. Cancer Cells 2,1-8. Wang JC (1996). DNA topo isomerases. Annual Review of Biochemistry 65, 635-692. 10 Zhang JG, Goldman JM, and Cross CNP (1995). Characterization of genomic BCR-ABL breakpoints in chronic myeloid leukaemia by PCR. British Journal of Haematology 90,138-146.
Three AF-IO signals (arrow), indicating a split of one YAC signal, and two centromere 10 signals (triangle) are detectable (panel A). One proximal (arrow) and one distal CALMYAC (triangle) are separated, indicating a breakpoint within this genomic region (panel B). Two MLL signals (arrow) and two centromere 11 signals (triangle) merely exclude a rearrangement of the MLL region (panel C). 1D-F. AMLwith AFlOand MLL Rearrangement. Three AF-IO signals (arrow), indicating a split of one YAC signal, and two centromere 10 signals (triangle) are detectable (panel D).
Chronic granulocytic leukemia is the most common leukemia observed after organ transplantation (22). Chronic myelomonocytic leukemia and acute myeloid leukemia following renal transplantation has also been reported [23,24) . Chronic granulocytic leukemia in the tran splanted 3. 4. 5. 6. 7. 8. Penn I. Tumours after renal and cardiac transplantation . Hematology/Oncology Clinics of North America, 7: 431-445, 1993. Penn I. Malignant neoplasia in the immunosuppressed pati ent. In Cooper DKC, Novitsky D eds.
Acute Leukemias VIII: Prognostic Factors and Treatment Strategies by P. D. Aplan, M. Stanulla (auth.), Prof. Dr. T. Büchner, Prof. Dr. med. W. Hiddemann, Prof. Dr. B. Wörmann, Prof. Dr. G. Schellong, Prof. Dr. J. Ritter, Prof. Dr. U. Creutzig (eds.)